Prostate cancer vs bph

prospectively Prostate cancer vs bph studied a sample Prostate cancer vs bph of men with Prostate cancer vs bph ED and smoking as their only risk factor; excluded were men with other risk factors for ED such as diabetes, hypertension, dyslipidemia, peripheral vascular disease, psychiatric disorders, and renal failure.

At baseline, severity of ED was found to be significantly correlated to duration of exposure in pack-years (32). At follow-up 1 year after smoking cessation, patients who successfully stopped smoking (ex-smokers) had a 25% improvement in erectile function, while men who continued (current smokers) did not improve. Additionally, a larger proportion of current smokers (7%) than ex-smokers (2.5%) had worsening of their baseline ED. This study suggests a large degree of Prostate cancer Prostate cancer treatment sloan kettering vs bph stabilization or improvement in ED after prostate cancer vs bph smoking cessation. These results were corroborated in a randomized prostate cancer vs bph controlled study of Chinese men enrolled in a nicotine replacement therapy (NRT) program with or without counseling. Six months after enrollment, patients who prostate cancer vs bph successfully quit smoking were more likely to have improvement in erectile function compared to persistent smokers (53.8% vs. 28.1%, P3,000 kcal/week significantly reduced the likelihood of severe ED (IIEF-5 Prostate cancer vs bph 600 mL/week) (6). Furthermore, in a large, multi-national epidemiologic study, heavy and no alcohol consumption were associated with higher risk of ED as prostate cancer vs bph compared to moderate alcohol intake (1 to 7 drinks per week), though not significantly (48). On the contrary, in the HPFS study, there was no change in relative risk of ED across all categories of alcohol consumption (8). In the rat model, chronic alcohol consumption leads to an upregulation of endothelin-1 (ET-1) which acts as a vasoconstrictor in the corpora cavernosa (CC).

Following electrical stimulation of the major pelvic ganglion, ethanol treated rats demonstrated significantly reduced erectile response as measured by maximal intracavernosal pressure/mean arterial pressure (ICP/MAP) (49).





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