Outlook If the disease is found before it prostate adenomatous spreads to other organs in a process known as metastasis, the 5-year survival rate is 99 percent. Once the cancer metastasizes, or spreads, the 5-year survival Prostate adenomatous rate is 29 percent. Regular screening can help detect prostate cancer while it is still treatable.
Risk factors The exact cause of prostate cancer is unclear, Prostate adenomatous but there are many possible risk factors. Age Prostate cancer is rare among men under the age of 45 years, but more common after the age of 50 years. Geography Prostate cancer occurs most frequently in North America, northwestern Europe, on the Caribbean islands, and in Australia. Genetic factors Certain genetic Prostate Adenomatous and ethnic groups have an increased risk of Prostate adenomatous prostate cancer. In the U. S., prostate cancer is prostate adenomatous at least 60 percent more common and 2 to 3 times more deadly among black men than non-Hispanic white men. A man also has a much higher risk of developing cancer if his identical twin has it, and a man whose brother or father had prostate cancer has twice the risk of developing it compared to other men. Having a brother who has or has had prostate cancer is more of a genetic risk than having a prostate adenomatous father with the disease.
Diet Studies have suggested that Prostate adenomatous a diet high in red meat or high-fat dairy products may increase a person's chances of developing prostate cancer, but the link is neither Prostate adenomatous confirmed nor clear.
Medication Some research has suggested that non-steroidal anti-inflammatory drug (NSAID) use may reduce the risk of prostate cancer. Others have linked NSAID use Prostate adenomatous with a higher risk of death from the disease. This is a controversial area, and results have not been confirmed. There has also been some investigation Prostate adenomatous into whether statins might slow the progression of prostate cancer.
One 2016 study concluded that results were "weak and inconsistent." Obesity It is often believed that obesity is linked to the development of Prostate Prostate adenomatous cancer, but the American Cancer Society maintains that there is no clear link. Some studies have found Prostate adenomatous that obesity increases the risk of death in advanced cancers. Studies have also concluded that obesity decreases Prostate adenomatous the risk that a cancer will be low-grade if it does occur. Agent Orange Exposure to Agent Orange, a chemical weapon used in the Vietnam war, prostate adenomatous may possibly be linked to the development of more aggressive types of cancer, but the extent of this has not been confirmed. This condition is known as Benign Prostatic Hyperplasia (or prostate adenoma) because it refers to the non-malignant growth of prostatic tissue Prostate adenomatous in men from middle age onwards. In a young man, the prostate gland is at an almost undetectable rudimentary stage, and spreads across the coating of the urinary tract like a cluster of seeds. However, during puberty, its form begins to change due to the increased production of the testosterone and Prostate adenomatous dihydrotestosterone hormones. Testosterone causes the "seeds", called acini, to Prostate adenomatous germinate and grow.
In adult life, due to this growth and proliferation, these small glandular structures can give rise to obstructive symptoms associated with urinary flow disorders. Benign Prostatic Hyperplasia is one of the most common diseases in men and is considered to be part of the physiological ageing process. The prostate adenomatous incidence of this disease is 51% in men aged 60 to 69, and between 75% and 90% in octogenarians. Symptomatology Although the definition of BPH encapsulates Prostate adenomatous the enlargement of the prostate and the mechanical Prostate adenomatous obstruction of the flow of urine, a direct relationship between the volume of hyperplasia and the intensity of symptoms is not always present. Very enlarged prostates do not always cause symptoms and other less prostate adenomatous enlarged organs can be a source of intense discomfort. The symptoms of Benign Prostatic Hyperplasia can be divided into two categories: obstructive and irritative. Obstructive symptoms Weak and/or intermittent flow of urine. Double voiding (urinating for a second time within a period of 2 hours). Irritative symptoms Polyuria, or increased frequency of urination. Nocturia, or the need to urinate frequently during the night.
Treatment of Benign Prostatic Hyperplasia Early prostate adenomatous diagnosis of Benign Prostatic Hyperplasia is essential to avoid complications such as urinary tract infections, damage to Prostate adenomatous the kidneys or bladder, bladder stones and incontinence. The Prostate adenomatous treatment for BPH will be determined by the potential for the existence of these secondary processes that derive from urinary obstruction, along with the patient’s level of discomfort Medical treatment In patients with mild Prostate adenomatous Prostate adenomatous or moderate symptoms, a pharmacological treatment can be administered that is based on alpha-blockers and alpha-reductase inhibitors, which can help to improve urinary flow. The former reduce the tension of the muscle cells inside the prostate, helping the patient to empty their bladder more effectively and comfortably. The latter reduce the size of the prostate, which improves the quality of prostate adenomatous urination. Surgical treatment Overview Image showing a normal prostate versus a prostate with a tumor Prostate cancer Prostate cancer is cancer that occurs in the Prostate adenomatous Prostate adenomatous prostate — a small walnut-shaped gland in men that prostate adenomatous produces the seminal fluid that nourishes and transports sperm.
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